By Hiroshi Mitsumoto MD
This crucial publication covers each element of residing with ALS, from medical positive aspects of the ailment to an summary of symptom administration to knowing its prognosis. significant sections take care of overseeing remedy and rehabilitation, residing with ALS, the disease’s complicated part, end-of-life matters, and assets. greatly revised and rewritten, this version includes new chapters at the most recent methods to treating ALS signs, using noninvasive ventilators, multidisciplinary group care, new instructions being built by means of the yankee Academy of Neurology for sufferers with ALS, using riluzole (Rilutek), and extra.
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Extra info for Amyotrophic Lateral Sclerosis: A Guide for Patients and Families
Drug Therapies The failure to find an effective drug therapy for ALS has not been for lack of trying. 2). Over the past several decades the treatments tested have covered the gamut from antiviral agents and antibiotics to chemotherapies, from immunomodulating agents to neurotrophic factors. What Is Amyotrophic Lateral Sclerosis? 2 Therapies Tried for Amyotrophic Lateral Sclerosis (Continued ) Neurotrophic agents Brain-derived neurotrophic factor (BDNF) Calcium channel blockers and chelating agents Calcium disodium edetate Ciliary neurotrophic factor (CNTF) Dantrolene Insulin-like growth factor (IGF-1, myotrophin) Nimodipine Xaliproden Verapamil Antimicrobial agents Other Ceftriaxone Adrenal cortex Tilerone hydrochloride Antihistaminic agents Trypan blue Trypan red Balneotherapy (natural mineral water baths) Antiviral agents Bovine gangliosides Amantadine Bromocriptine Indinavir Isoprinosine Cervico-dorsal electroshock therapy Transfer factor Guanidine Zidovudine 5-hydroxytrytophan Nutritional supplements and vitamins Hyperbaric oxygen Crude liver extract Phthalazinol Intravenous amino acids Octacosanol Snake venom (modified neurotoxin) Vitamin B1 (thiamine) Stem cells Vitamin B6 (pyridoxine) Toluloxy propanediol Penicillamine Olfactory cells Vitamin B12 “Vitamin B” in yeast preparation Probably the most ubiquitous treatment for ALS over the past century has been the antioxidant vitamin E (tocopherol).
When dexterity is impaired, voluntary and even reflex motions become awkward. Patients may initially experience stiffness, slowness, and clumsiness when performing any motion that requires fine movement, such as any rapid repetitive motion of the fingers, feet, and even the lips or tongue. Loss of Muscle Strength (Weakness) Another sign of upper motor neuron dysfunction is a loss of muscle strength. Because the upper motor neurons are affected but lower motor units are normally preserved, muscle weakness resulting from upper motor neuron dysfunction generally is mild and not as severe as that seen in lower motor neuron involvement.
Loss of Muscle Strength (Weakness) Another sign of upper motor neuron dysfunction is a loss of muscle strength. Because the upper motor neurons are affected but lower motor units are normally preserved, muscle weakness resulting from upper motor neuron dysfunction generally is mild and not as severe as that seen in lower motor neuron involvement. In patients with severe upper motor neuron disease, accurately assessing the muscle strength becomes difficult because muscle spasticity and loss of dexterity prevent effective activation of the motor units.
Amyotrophic Lateral Sclerosis: A Guide for Patients and Families by Hiroshi Mitsumoto MD